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thioredoxin reductase 2, mitochondrial; selenoprotein Z; selZ; TR-beta; thioredoxin reductase TR3 (TXNRD2, KIAA1652, TRXR2)

Function: - maintains thioredoxin in a reduced state - implicated in the defenses against oxidative stress - may play a role in redox-regulated cell signaling - the active site is a redox-active disulfide bond - the selenocysteine residue is essential for enzymatic activity thioredoxin + NADP+ = thioredoxin disulfide + NADPH Cofactor: FAD (putative) Structure: - homodimer - belongs to the class-I pyridine nucleotide-disulfide oxidoreductase family Compartment: mitochondria Alternative splicing: named isoforms=4 Expression: - highly expressed in the prostate, ovary, liver, testis, uterus, colon & small intestine - moderately expressed in brain, skeletal muscle, heart & spleen - low expression in placenta, pancreas, thymus & peripheral blood leukocytes - high levels in kidney [2]; low levels in kidney [3]

General

flavoprotein mitochondrial protein oxidoreductase selenoprotein

Properties

SIZE: entity length = 524 aa MW = 57 kD COMPARTMENT: mitochondria MOTIF: cofactor-binding site [41-70] COFACTOR-BOUND: flavin adenine dinucleotide cysteine residue {C86} MODIFICATION: cysteine residue {C91} cysteine residue {C91} MODIFICATION: cysteine residue {C86} histidine residue {H497} selenocysteine residue {C523}

Database Correlations

OMIM 606448 UniProt Q9NNW7 PFAM correlations Entrez Gene 10587 Kegg hsa:10587 ENZYME 1.8.1.9

References

  1. UniProt :accession Q9NNW7
  2. Lescure A et al Novel selenoproteins identified in silico and in vivo by using a conserved RNA structural motif. J Biol Chem. 1999 Dec 31;274(53):38147-54. PMID: 10608886
  3. Gasdaska PY Cloning, sequencing and functional expression of a novel human thioredoxin reductase FEBS Lett. 1999 Jan 8;442(1):105-11. PMID: 9923614