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hyperlipoproteinemia type 1; familial hyperchylomicronemia

Etiology: 1) primary or familial (see types 1a & 1b) 2) secondary forms (transient) a) acute pancreatitis b) uncontrolled diabetes mellitus c) hypothyroidism d) dysglobulinemia e) systemic lupus erythematosus f) oral contraceptives Epidemiology: 1) extremely uncommon 2) familial forms diagnosed < 10 years of age 3) secondary forms in adults Pathology: 1) deficiency in lipoprotein lipase, or 2) deficiency in apolipoprotein C2 3) increased plasma chylomicrons Genetics: - recessve form associated with defect in lipoprotein lipase - associated with defects in USF1 Clinical manifestations: 1) recurrent abdominal pain 2) hepatosplenomegaly 3) eruptive xanthomas 4) lipemia retinalis 5) acute pancreatitis 6) peritonitis 7) manifests in childhood Laboratory: 1) examination of the specimen for a creamy layer (evidence of chylomicrons) 2) fasting lipid panel -> increased triglycerides (chylomicrons) 3) serum amylase a) may be falsely normal due to an unidentified inhibitory factor b) dilution of the serum with normal saline may increase amylase levels Management: 1) major objective is to prevent recurrent abdominal episodes 2) strict adherence to diet a) total fat < 0.5. mg/kg/day b) medium-chain triglycerides that enter portal venous system directly without formation of chylomicrons may be used if adequate calories is an issue 3) acute abdominal attack a) intravenous therapy for 1st 24 hours b) totally fat-free diet until resolution

Related

acute pancreatitis apolipoprotein C2; apo-CII (APOC2, APC2) chylomicron diabetes mellitus eruptive xanthoma; eruptive xanthelasma hypothyroidism lipoprotein lipase (lipase-D, post heparin lipase, LPL, LIPD) oral contraceptive (OC) systemic lupus erythematosus

Specific

hyperlipoproteinemia type 1A hyperlipoproteinemia type 1B

General

hyperlipoproteinemia (HLP) lipid metabolism, inborn error; lipid storage disease; lipidosis

Figures/Diagrams

Assessment of lipoprotein disorders

References

  1. Clinical Diagnosis & Management by Laboratory Methods, J.B. Henry (ed), W.B. Saunders Co., Philadelphia, PA. 1991, pg 207
  2. Tietz Textbook of Clinical Chemistry, 2nd ed. Burtis CA & Ashwood ER (eds), WB Saunders Co, Philadelphia PA, 1993, pg 1035