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hydrolase PNKD (paroxysmal nonkinesiogenic dyskinesia protein, myofibrillogenesis regulator 1, MR-1, trans-activated by hepatitis C virus core protein 2, PNKD, KIAA1184, MR1, TAHCCP2, FKSG19, UNQ2491/PRO5778)

Function: - probable hydrolase - plays an aggravative role in the development of cardiac hypertrophy via activation of the NF-kappa-B signaling pathway (putative) - isoform 2 interacts with the sarcomeric proteins, MRLC2, MYOM1 & ENO3 - isoform 2 is phosphorylated at Ser-121 upon DNA damage, probably by ATM or ATR Structure: - belongs to the metallo-beta-lactamase superfamily, glyoxalase II family Compartment: - isoform 1: membrane, peripheral membrane - isoform 2: cytoplasm, nucleus - isoform 3: mitochondrion Alternative splicing: named isoforms=4 Expression: - isoform 1 is only expressed in the brain - isoform 2 is ubiquitously detected with highest expression in skeletal muscle & detected in myocardial myofibrils - variant Val-7 & Val-9 are detected in the brain only - induced by Hepatitis C virus core protein Pathology: - defects in PNKD are a cause of paroxysmal nonkinesiogenic dyskinesia type-1 Laboratory: - PNKD gene mutation

General

hydrolase metalloprotein phosphoprotein

Properties

SIZE: entity length = 385 aa MW = 43 kD MOTIF: Ser phosphorylation site {121-121} Zn+2-binding site SITE: 172-172 Zn+2-binding site SITE: 174-174 Zn+2-binding site SITE: 176-176 Zn+2-binding site SITE: 177-177 Zn+2-binding site SITE: 229-229 Zn+2-binding site SITE: 253-253 Substrate {291-293} MOTIF: Zn+2-binding site SITE: 291-291 Substrate {376-379}

Database Correlations

OMIM 118800 UniProt Q8N490 Pfam PF00753

References

  1. UniProt :accession Q8N490
  2. GeneReviews https://www.genecards.org/cgi-bin/carddisp.pl?gene=PNKD