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fumarase (fumarate hydratase mitochondrial, FH)

Function: - catalyzes conversion of fumarate to malate - carbohydrate metabolism, tricarboxylic acid cycle - acts as a tumor suppressor (S)-malate fumarate + H2O Structure: - homotetramer - belongs to the class-II fumarase/aspartase family, fumarase subfamily - initiator Met-1 is removed - contains a N-acetylalanine at position 2 - isoform cytoplasmic is acetylated at position 2 - 2 substrate binding sites: a) catalytic A site b) non-catalytic B site may play a role in transfer of substrate or product between active site & solvent; alternatively, the B site may bind allosteric effectors Compartment: mitochondria, cytoplasm Alternative initiation: named isoforms=2 Pathology: - defects in FH are the cause of fumarase deficiency - defects in FH are the cause of multiple cutaneous & uterine leiomyomata - defects in FH are the cause of hereditary leiomyomatosis & renal cell cancer

Interactions

molecular events

Related

citric acid cycle, Krebs cycle or tricarboxylic acid [TCA] cycle

General

citric acid cycle enzyme hydro-lyase, hydratase or dehydratase

Properties

SIZE: entity length = 510 aa MW = 55 kD COMPARTMENT: mitochondria cytoplasm MOTIF: binding site SITE: 147-147 FOR-BINDING-OF: Substrate B site {176-179} Substrate binding {186-188}

Database Correlations

OMIM correlations MORBIDMAP 136850 UniProt P07954 Pfam PF00206 ENZYME 4.2.1.2

References

  1. UniProt :accession P07954
  2. Atlas of Genetics & cytogenetics in oncology & Haematology http://atlasgeneticsoncology.org/Genes/FHID40573ch1q42.html
  3. GeneReviews https://www.genecards.org/cgi-bin/carddisp.pl?gene=FH