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ectodysplasin-A; ectodermal dysplasia protein; EDA protein; contains: ectodysplasin-A, membrane form; contains: ectodysplasin-A, secreted form (EDA, ED1, EDA2)

Function: - seems to be involved in epithelial-mesenchymal signaling during morphogenesis of ectodermal organs - isoform 1 binds only to the receptor EDAR, while isoform 3 binds exclusively to the receptor XEDAR - processing by furin produces a secreted form - homotrimer - homotrimers may then dimerize & form higher order oligomers Structure: - N-glycosylated - belongs to the tumor necrosis factor family - contains 1 collagen-like domain Compartment: - cell membrane; single-pass type 2 membrane protein - ectodysplasin-A, secreted form: secreted Alternative splicing: - named isoforms=8 - additional isoforms seem to exist - at least one isoform may be produced at very low levels due to a premature stop codon in the mRNA, leading to nonsense-mediated mRNA decay Expression: - not abundant - expressed in specific cell types of ectodermal (but not mesodermal) origin of keratinocytes, hair follicles, sweat glands - also expressed in adult heart, liver, muscle, pancreas, prostate, fetal liver, uterus, small intestine & umbilical chord Pathology: - defects in EDA are the cause of a) ectodermal dysplasia type 1 b) tooth agenesis selective X-linked type 1

Related

anhidrotic ectodermal dysplasia tumor necrosis factor receptor superfamily member EDAR; Anhidrotic ectodysplasin receptor 1; ectodysplasin-A receptor; EDA-A1 receptor; ectodermal dysplasia receptor; downless homolog (EDAR, DL)

General

glycoprotein membrane protein

Properties

SIZE: entity length = 391 aa MW = 41 kD COMPARTMENT: cellular membrane MOTIF: transmembrane domain {42-62} proteolytic site {159-160} Collagen-like {180-229} N-glycosylation site {N313} N-glycosylation site {N372}

Database Correlations

OMIM correlations UniProt Q92838 PFAM correlations Entrez Gene 1896 Kegg hsa:1896

References

  1. UniProt :accession Q92838
  2. GeneReviews http://www.ncbi.nlm.nih.gov/sites/genetests/lab/gene/EDA