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acrodermatitis enteropathica

Etiology: 1) zinc deficiency - Crohn's disease with or without ileal resection - small intestinal disease [6] 2) inherited 3) acquired Epidemiology: rare Pathology: - inability to absorb sufficient Zn+2 - immune system dysfunction - skin & skin appendage pathology Genetics: - autosomal recessive - associated with defects in SLC39A4 Clinical manifestations: 1) growth retardation 1) angular cheilitis 2) seborrheic dermatitis 3) skin erosions, blisters, pustules 4) erythematous vesiculobullous dermatitis especially involving a) face b) hands c) feet d) perineum 5) alopecia 6) diarrhea 7) occasionally mental disorders * images [3,4,5] Management: 1) zinc supplementation 3 mg/kg 2) without Zn+2 therapy, disease is fatal

Interactions

disease interactions

General

acrodermatitis disorder of zinc metabolism genetic disease of the gastrointestinal tract genetic disease of the skin (genodermatosis)

Database Correlations

OMIM 201100

References

  1. Mayo Internal Medicine Board Review, 1998-99, Prakash UBS (ed) Lippincott-Raven, Philadelphia, 1998, pg 174
  2. OMIM :accession 201100
  3. DermNet NZ. Acrodermatitis enteropathica (images) http://www.dermnetnz.org/systemic/acrodermatitis-enteropathica.html
  4. Sivakumar A, Vageshappa RK, Kumari R. Acrodermatitis Enteropathica. JAMA Dermatol. Published online November 8, 2023 PMID: 37938848 https://jamanetwork.com/journals/jamadermatology/fullarticle/2811789
  5. Guliani A, Bishnoi A. Acquired Acrodermatitis Enteropathica. JAMA Dermatol. 2019 Nov 1;155(11):1305. PMID: 31461117 https://jamanetwork.com/journals/jamadermatology/fullarticle/2747827
  6. NEJM Knowledge+