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trichothiodystrophy (TTD)

Pathology: - cells from individuals with photosensitive TTD have defects in nucleotide excision repair indistinguishable from cells obtained from patients with Xeroderma Pigmentosum (XP) - tiger tail banding of hair strands [2]* * image [2] Genetics: 1) autosomal recessive 2) associated with defects in GTF2H5 gene 3) associated with defects in TTDN1 gene (non-photosensitive) 4) associated with defects in ERCC2, ERCC3 (photosensitive) Clinical manifestations: 1) sulfur-deficient brittle hair 2) small size, impaired sexual development 3) mental retardation 5) unusual facial features 6) photosensitivity (frequent) 7) ichthyosis (fish-like scales on skin) a) neonates with trichothiodystrophy & ichthyosis are usually born with a collodion membrane b) severity of the ichthyosis after the membrane is shed is variable, ranging from a mild to severe 8) newborn withe frizzy hair, low set ears [2]* * image [2] Complications: - individuals with TTD do not appear to have an increased risk of malignant neoplasms

Related

nucleotide excision repair

General

dwarfism (nanism) progeroid syndrome

References

  1. Lehmann AR. Nucleotide excision repair and the link with transcription. Trends Biochem Sci. 1995 Oct;20(10):402-5. Review. PMID: 8533152
  2. Garon L, Kokta V, Coulombe J. Images in Dermatology Trichothiodystrophy JAMA Dermatol. 2023;159(8):877 PMID: 37342013 https://jamanetwork.com/journals/jamadermatology/fullarticle/2806332

Databases & Figures

OMIM correlations Human Disorders of DNA Repair