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progressive supranuclear palsy; Steele-Richardson-Olszewski syndrome (PSP)

Parkinsonism plus disturbances of ocular motility. Epidemiology: 1) rare (4% of patients with parkinsonism) 2) age of onset: after age 50, generally 60-70 years Pathology: 1) aberration of tau protein - abnormal staining in pretangles/neurofibrillary tangles - tuft shaped astrocytes 2) degenerative changes in: - brainstem, midbrain (substantia nigra), cerebellum, nucleus basalis, cerebral cortex, diencephalon, basal ganglia (globus pallidus) 3) neuronal loss 4) gliosis 5) neurofibrillary tangles (different from Alzheimer's disease) 6) iron accumulation in brain - astrocytes accumulate iron & iron deposition is found in regions with tau deposition [11] - hemoglobin & neuroglobin are dysregulated in these same brain regions [11] Genetics: - associated with polymorphism (Arg-7) in saitohin gene - subhaplotypes of MAPT associated with risk of PSP & severity of tau pathology [10] Clinical manifestations: 1) supranuclear vertical ophthalmoplegia a) inability to move the eyes vertically b) not always 1st sign; generally present by year 4 c) downward gaze affected more than upward gaze 2) other oculomotor & eyelid disturbances a) wide-eyed b) square-wave jerks (fixation instability) c) intact Doll's eye movement 3) gait ataxia, postural instability - early falls (often backward) 4) parkinsonism a) axial rigidity > limb rigidity b) bradykinesia c) resting tremor (often not prominent) 5) dysarthria, slurred speech 6) dysphagia 7) nuchal extension & rigidity (hyperextension of neck) 8) facial dystonia: - contracted facial muscles may give the appearance of sustained surprise [6] 9) pyramidal tract signs 10) cognitive or behavioral changes a) dementia - slowed processing speed - impaired executive function b) emotional lability c) apathy 11) hypertension may be present 12) late stage: immobility 13) marked incapacity within 3-5 years Laboratory: - generally normal - higher levels of neurofilament light chain in serum in multiple system atrophy or progressive supranuclear palsy than in Parkinson's disease & healthy controls Radiology: 1) magnetic resonance imaging & CT may be normal or may show frontal &/or midbrain atrophy 2) PET scan is generally shows frontal hypometabolism (2-fluoro-2-deoxyglucose), but may be normal Complications: - complications of immobility Management: 1) poor response to Sinemet 2) control of agitation, emotional lability - selective serotonin reuptake inhibitor (SSRI) 3) local botulinum toxin A (Botox) may improve rigidity 4) selective serotonin reuptake inhibitors (SSRI) may improve dysarthria & dysphagia [4] 5) electroconvulsive therapy of limited benefit for improving bradykinesia, rigidity, gait instability 6) avoid neuroleptics 7) prognosis: a) marked incapacity in 3-5 years b) death < 10 years from complication of immobility Clinical trials: - davunetide is not effective [9]

Interactions

disease interactions

General

brainstem disease neurodegenerative disease parkinson plus syndrome

Properties

PATHOLOGY: neurofibrillary tangle SITE: hippocampus basal ganglia brainstem

Database Correlations

OMIM correlations

References

  1. Lang AE & Lazano AM Parkinson's disease. First of two parts. NEJM 339(15):1044-53 1998 PMID: 9761807
  2. Bronstein J, In: Intensive Course in Geriatric Medicine & Board Review, Marina Del Ray, CA, Sept 29-Oct 2, 2004
  3. Miller B, UCSF Memory & Aging Center, 2001
  4. Handforth CA, WLA VA Dept of Neurology {patient-related communication}
  5. Geriatrics Review Syllabus, American Geriatrics Society, 5th edition, 2002-2004; 7th edition 2010
  6. Medical Knowledge Self Assessment Program (MKSAP) 14, 16. American College of Physicians, Philadelphia 2006, 2012
  7. Messina D, Cerasa A, Condino F et al Patterns of brain atrophy in Parkinson's disease, progressive supranuclear palsy and multiple system atrophy. Parkinsonism Relat Disord. 2011 Mar;17(3):172-6 PMID: 21236720
  8. Wenning GK, Colosimo C. Diagnostic criteria for multiple system atrophy and progressive supranuclear palsy. Rev Neurol (Paris). 2010 Oct;166(10):829-33 PMID: 20813385
  9. Boxer AL et al. Davunetide in patients with progressive supranuclear palsy: A randomised, double-blind, placebo-controlled phase 2/3 trial. Lancet Neurol 2014 Jul; 13:676 PMID: 24873720 http://www.thelancet.com/journals/laneur/article/PIIS1474-4422(14)70088-2/fulltext
  10. Heckman MG, Brennan RR, Labbe C et al Association of MAPT Subhaplotypes With Risk of Progressive Supranuclear Palsy and Severity of Tau Pathology. JAMA Neurol. Published online March 18, 2019. PMID: 30882841 https://jamanetwork.com/journals/jamaneurology/fullarticle/2728174
  11. Lee S et al. Cell-specific dysregulation of iron and oxygen homeostasis as a novel pathophysiology in PSP. Ann Neurol 2022 Oct 30; [e-pub]. PMID: 36309960 https://onlinelibrary.wiley.com/doi/10.1002/ana.26540
  12. NINDS Progressive Supranuclear Palsy Information Page https://www.ninds.nih.gov/Disorders/All-Disorders/Progressive-Supranuclear-Palsy-Information-Page