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pancuronium (Pavulon)

Tradename: Pavulon. (pancuronium bromide) Indications: - relaxation of skeletal muscles during surgery under general anesthesia - relaxation of skeletal muscles during endotracheal intubation - increase pulmonary compliance during mechanical ventilation - status asthmaticus resistant to other therapy - neuromuscular blocking agent of choice in most critically ill patients WITHOUT significant cardiac disease or hemodynamic instability Dosage: 1) ALWAYS use with sedative, i.e. lorazepam (Ativan) 2) should be used with analgesic if needed 3) start: 0.06-0.1 mg/kg/hr (4-5 mg) maintenance: 0.02-0.03 mg/kg (1-2 mg/hr) 4) 0.04 mg/kg followed by 0.02-0.1 mg/kg every 30-60 min PRN 5) titrate dose to patient's needs Injection: 1 mg/mL (10 mL), 2 mg/mL (2 mL, 5 mL). Dosage adjustment in renal failure: creatinine clearance dosage 10-50 mL/min 50% of normal dose < 10 mL/min do NOT use Phamacokinetics: 1) rapid onset of action (4 minutes) 2) onset & duration of action are dose-dependent 3) 1/2life is 2 hours 4) duration 45-90 min 5) metabolized in liver 30-40% to several compounds, some which are active 6) eliminated mainly in the urine (55-70% as unchanged drug) 7) 1/2life is prolonged in patients with hepatic or renal impairment Adverse effects: 1) not common (1-10%) - rash, excessive salivation, itching, tachycardia, hypertension (secondary to vagolytic effect) 2) uncommon (< 1%) - skin flushing, erythema, edema, circulatory collapse, bronchospasm, hypersensitivity, burning sensation along the vein, profound muscle weakness, wheezing 3) other - mast cell degranulation & histamine release - allergic-type reaction - rash - increased risk of deep vein thrombosis (DVT) - prolonged paralysis* - exacerbation of adverse effects with hyperkalemia or hypokalemia * Cholinesterase inhibitors (neostigmine, pyridostigmine, edrophonium) are used with atropine to REVERSE neuromuscular blockade Drug interactions: 1) aminoglycosides, tetracyclines, clindamycin, K+-depleting agents (loop diuretics, thiazides, amphotericin B, corticosteroids) in combination increase & prolong neuromuscular blockade 2) corticosteroids in combination prolong muscle weakness 3) quinidine 4) magnesium sulfate 5) verapamil Laboratory: 1) specimen: a) plasma b) stable for 20 hours at 23 degrees C, pH 5.4 c) unstable at room temperature, pH 7.4 2) methods: fluorometry, GC 3) labs with Loincs - pancuronium in tissue - pancuronium in blood - pancuronium in serum/plasma - pancuronium in urine Mechanism of action: 1) non-depolarizing neuromuscular blocking agent 2) diminishes response to acetylcholine at the motor endplate

Interactions

drug interactions

General

neuromuscular blocker (paralyzing agent) steroid

Properties

MISC-INFO: elimination route LIVER KIDNEY 1/2life 2 HOURS therapeutic-range >0.2 UG/ML protein-binding 7-29% pregnancy-category C safety in lactation ?

Database Correlations

PUBCHEM correlations

References

  1. Goodman and Gilman's The Pharmacological Basis of Therapeutics, 8th ed. Gilman et al, eds. Permagon Press/McGraw Hill, 1990, pg 169
  2. Drug Information & Medication Formulary, Veterans Affairs, Central California Health Care System, 1st ed., Ravnan et al eds, 1998 - not on National VA formulary
  3. Kaiser Permanente Northern California Regional Drug Formulary, 1998
  4. Clinical Guide to Laboratory Tests, 3rd ed. Teitz ed., W.B. Saunders, 1995
  5. Deprecated Reference