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NF-kappa B p52 subunit; NF-kappa p100; DNA-binding factor KBF2; H2TF1; lymphocyte translocation chromosome 10; oncogene Lyt-10; Lyt10 (NFKB2, LYT10)

Function: - p100 cleaved to form p52 & p49 - in a non-canonical activation pathway, the MAP3K14-activated CHUK/IKKA homodimer phosphorylates NFKB2/p100 associated with RelB, inducing its proteolytic processing to NFKB2/p52 & formation of NF-kappa-B RelB-p52 complexes - NF-kappa-B heterodimeric RelB-p52 complex is a transcriptional activator - NF-kappa-B p52-p52 homodimer is a transcriptional repressor - NFKB2 - appears to have dual functions such as cytoplasmic retention of attached NF-kappa-B proteins by p100 & generation of p52 by cotranslational processing - proteasome-mediated process ensures production of both p52 & p100 & preserves their independent function - p52 binds to the kappa-B consensus sequence 5'-GGRNNYYCC-3', located in the enhancer region of genes involved in immune response & acute phase reactions - p52 & p100 are respectively the minor & major form; the processing of p100 being relatively poor - isoform p49 is a subunit of the NF-kappa-B protein complex, which stimulates the HIV enhancer in synergy with p65 - component of the NF-kappa-B RelB-p52 complex - component of the NF-kappa-B p52-p52 complex - component of the NF-kappa-B p65-p52 complex - component of the NF-kappa-B p52-c-Rel complex - NFKB2/p52 interacts with NFKBIE - component of a complex consisting of the NF-kappa-B p50-p50 homodimer & BCL3 - while translation occurs, the particular unfolded structure after the GRR repeat promotes the generation of p52 making it an acceptable substrate for the proteasome (cotranslational processing; the processed form is active & the unprocessed form acts as an inhibitor (I kappa B-like), being able to form cytosolic complexes with NF-kappa B, trapping it in the cytoplasm - complete folding of the region downstream of the GRR repeat precludes processing subsequent to MAP3K14-dependent serine phosphorylation - p100 polyubiquitination occurs then triggering its proteasome-dependent processing - constitutive processing is tightly suppressed by its C-terminal processing inhibitory domain, named PID, which - contains the death domain Structure: - homodimer - heterodimer with other NF kappa B protein - C-terminus of p100 might be involved in cytoplasmic retention, inhibition of DNA-binding by p52 homodimers, &/or transcription activation (putative) - glycine-rich region (GRR) appears to be a critical element in the generation of p52 - contains 7 ANK repeats - contains 1 death domain - contains 1 RHD (Rel-like) domain Compartment: - nucleus, cytoplasm - nuclear, but also found in the cytoplasm in an inactive form complexed to an inhibitor (I-kappa-B) Alternative splicing: named isoforms=2 Pathology: - chromosomal translocation t(10;14)(q24;q32) involving NFKB2 with IGHA1 is found in a case of B-cell non Hodgkin lymphoma, the resulting oncogene is also called Lyt-10C alpha variant - chromosomal aberration involving NFKB2 is found in a cutaneous T-cell leukemia (C-TCL) cell line; this rearrangement produces the p80HT gene which encodes for a truncated 80 kD protein (p80HT) - heterogeneous chromosomal aberrations founs in B-cell leukemia (B-CLL) cell lines, LB40 & EB308

General

oligomerizing protein phosphoprotein rel protein family

Properties

SIZE: entity length = 900 aa MW = 97 kD COMPARTMENT: cytoplasm cell nucleus MOTIF: RHD {38-343} MOTIF: nuclear translocation signal {337-341} GRR {346-377} glycine-rich region {350-400} Cleavage {454-455} ankyrin repeat NAME: ankyrin repeat SITE: 487-519 MOTIF: Breakpoint {490-491} ankyrin repeat NAME: ankyrin repeat SITE: 526-555 ankyrin repeat NAME: ankyrin repeat SITE: 559-591 ankyrin repeat NAME: ankyrin repeat SITE: 599-628 ankyrin repeat NAME: ankyrin repeat SITE: 633-663 ankyrin repeat NAME: ankyrin repeat SITE: 667-696 Ser phosphorylation site {S713} Ser phosphorylation site {S715} Ser phosphorylation site {S717} ankyrin repeat NAME: ankyrin repeat SITE: 729-758 death domain NAME: death domain SITE: 764-851 Ser phosphorylation site {S866} Ser phosphorylation site {S870}

Database Correlations

OMIM 164012 UniProt Q00653 PFAM correlations Entrez Gene 4791 Kegg hsa:4791

References

  1. UniProt :accession Q00653
  2. Entrez Gene :accession 4791
  3. Atlas of genetics & cytogenetics in oncology & haematology http://atlasgeneticsoncology.org/genes/NFKB2ID362.html