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lissencephaly X-linked type 1 (LISX1)

classic lissencephaly Pathology: - affected boys show an abnormally thick cortex with absent or severely reduced gyri Genetics: - defects associated with defects in DCX - t(X;2)(q22.3;p25.1) - female patients display a less severe phenotype referred to as "doublecortex" Clinical manifestations: - mental retardation - seizures that are more severe in male patients - feeding problems - abnormal muscular tone - severe to profound psychomotor retardation

Related

double cortex/X-linked lissencephaly syndrome; X-linked subcortical laminar heterotopia/lissencephaly syndrome; subcortical band heterotopia X-linked

General

lissencephaly; agyria

Database Correlations

OMIM 300067

References

OMIM :accession 300067