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leukocytoclastic vasculitis; hypersensitivity vasculitis; trisymptome

Etiology: 1) autoimmune disease - systemic lupus erythematosus 2) recent viral infection - hepatitis C, cryoglobulinemia 3) malignancy - lymphoproliferative disorders 4) drug adverse effect 5) systemic vasculitis 6) idiopathic (40%) [1] Clinical manifestations: - palpable purpura - cutaneous vesicles, pustules, maculopapular lesions, urticaria - occurs within 2 weeks of exposure to offending agent - generally resolves when the offending agent is removed [1] Laboratory: skin biopsy Differential diagnosis: - exanthematous drug eruption (morbilliform exanthem) Management: 1) generally self-limited - withdrawal of offending agent 2) glucocorticoids 3) see acute leukocytoclastic vasculitis (Henoch-Schonlein purpura) or specific etiology

Interactions

disease interactions

Related

cryoglobulinemia (cryoglobulinemic vasculitis) leukocytoclasis serum sickness; serum sickness-like reaction

Specific

acute leukocytoclastic vasculitis (Henoch-Schonlein purpura; IgA vasculitis) chronic leukocytoclastic vasculitis (Gougerot-Ruiter type) cutaneous leukocytoclastic vasculitis urticarial vasculitis

General

type 3 hypersensitivity; immune complex hypersensitivity vasculitis

References

  1. Medical Knowledge Self Assessment Program (MKSAP) 14, 16, 17, 18, 19. American College of Physicians, Philadelphia 2006, 2012, 2015, 2018. 2022.
  2. Eastham ABW, Diamond HS (images) Medscape: Leukocytoclastic Vasculitis. http://emedicine.medscape.com/article/333891-overview
  3. Kroshinsky D Adverse cutaneous reactions to medications Clin Dermatol. 2020 Nov-Dec;38(6):605-606 PMID: 33341194