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inwardly-rectifying K+ channel J1; ATP-sensitive inward rectifier K+ channel 1; K+ channel, inwardly rectifying subfamily J member 1; ATP-regulated K+ channel ROM-K; Kir1.1 (KCNJ1, ROMK1)

Function: - inwardly-rectifying K+ channel - in the kidney, probably plays a major role in K+ homeostasis - channel is activated by internal ATP & can be blocked by external Ba+2 Structure: - belongs to the inward rectifier-type K+ channel family Compartment: membrane Alternative splicing: named isoforms=3 Expression: - expressed kidney & pancreatic islets - lower levels in skeletal muscle, pancreas, spleen, brain, heart & liver Pathology: - defects in KCNJ1 are the cause of Bartter syndrome type-2

General

ATP-sensitive K+ channel (inwardly-rectifying K+ channel) inwardly-rectifying K+ channel subfamily J

Properties

SIZE: entity length = 391 aa MW = 45 kD COMPARTMENT: plasma membrane CELL: beta cell WITHIN: islets of Langerhans ORGANISM: vertebrate* MOTIF: cytoplasmic domain {1-77} transmembrane domain {78-102} exoplasmic loop {103-155} MOTIF: H5 (pore-forming helix) {128-139} peptide motif {141-146} transmembrane domain {156-177} MOTIF: asparagine residue {171} cytoplasmic domain {178-391} MOTIF: ATP-binding site NAME: ATP-binding site SITE: 223-230 ION-PERMEABILITY: K+

Database Correlations

OMIM correlations MORBIDMAP 600359 UniProt P48048 Pfam PF01007 Entrez Gene 3758 Kegg hsa:3758

References

  1. UniProt :accession P48048
  2. GeneReviews http://www.ncbi.nlm.nih.gov/sites/genetests/lab/gene/KCNJ1