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CD152 (cytotoxic T-cell protein 4-1, cytotoxic T lymphocyte-associated serine esterase-4, CTLA-4, CD152 antigen, CTLA4, CD152)

Function: - role in T-cell activation - interacts with CD80 & CD86 to down regulate T cell activation - a checkpoint inhibitor - part of an immunological network that leads to tolerance - ligands: CD80, CD86, phosphatidylinositol-3-kinase protein tyrosine phsophatase 1D Structure: - significant homology to CD28 in the extracellular region Compartment: membrane Expression: - widely expressed with highest levels in lymphoid tissues - activated T cells & activated B cells - transcription regulated by: - AP1, CD28re, IL-2 kappa b, OCT-1 Pathology: - activated T cells have two transcripts with alternative poly- adenylation sites, the longer transcript contains a repeated -AU- motif, the length of which varies between different individuals, leading to a restriction fragment length polymorphism (RFLP) which has been linked to various human autoimmune diseases - genetic variation in CTLA4 influences susceptibility to systemic lupus erythematosus - genetic variation in CTLA4 may be a cause of susceptibility to Graves disease - genetic variation in CTLA4 is the cause of susceptibility to insulin-dependent diabetes mellitus type 12 - genetic variation in CTLA4 is the cause of susceptibility to celiac disease type 3 - genetic variations in CTLA4 are associated with susceptibility to hepatitis B virus infection Pharmacology: - monoclonal antibody to CTLA4, ipilimumab (Yervoy), FDA-approved to treat metastatic melanoma - monoclonal antibody to CTLA4, tremelimumab, in phase 3 clinical trials for treatment of metastatic melanoma Comparative biology: - anti-CTLA4 therapy can result in development of antibodies to gut flora - anti-CTLA4 therapy decreases numbers of Bacteroidales & Burkholderiales species & increases Clostridiales species in feces of mice

General

cluster-of-differentiation antigen; cluster designation antigen; CD antigen glycoprotein immunoglobulin superfamily protein serine protease

Properties

SIZE: entity length = 223 aa MW = 25 kD COMPARTMENT: cellular membrane CELL: cytotoxic T cell MOTIF: signal sequence {1-35} immunoglobulin superfamily domain {39-140} MOTIF: cysteine residue {C58} MODIFICATION: cysteine residue {C129} cysteine residue {C85} MODIFICATION: cysteine residue {C103} cysteine residue {C103} MODIFICATION: cysteine residue {C85} N-glycosylation site {N113} cysteine residue {C129} MODIFICATION: cysteine residue {C58} transmembrane domain {162-182}

Database Correlations

OMIM correlations MORBIDMAP 123890 UniProt P16410 Pfam PF07686 Kegg hsa:1493

References

  1. OMIM :accession 123890
  2. UniProt :accession P16410
  3. Protein Reviews on the Web http://mpr.nci.nih.gov/prow/guide/1686349937_g.htm
  4. http://www.pathologyoutlines.com/cdmarkers.html 20 May 2003
  5. Wikipedia; Note: CLTA-4 entry http://en.wikipedia.org/wiki/CTLA-4
  6. Vetizou M, Pitt JM, Daillere R et al Anticancer immunotherapy by CTLA-4 blockade relies on the gut microbiota. Science. 2015 Nov 27;350(6264):1079-84 PMID: 26541610